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lentiviral particles expressing shrna against murine glut3  (Santa Cruz Biotechnology)
92
Santa Cruz Biotechnology lentiviral particles expressing shrna against murine glut3
Fig. 7 Model describing how RIP140 inhibits tumorigenesis by affecting glycolysis through the blockade of <t>GLUT3</t> expres- sion. RIP140 and p53 cooper- ate to inhibit the expression of GLUT3 induced by HIF-α. Glycolysis-dependent prolif- eration of breast cancer cells is reduced, due to a decrease in glycolysis. The prognostic value of RIP140 is associated with good survival in patients with low GLUT3, high p53 and low HIF-α (left panel). In patients with high HIF-α, low p53 and high GLUT3, RIP140 and p53 do not inhibit the tran- scriptional activity of HIF-α; GLUT3 is highly expressed and glycolysis is enhanced. In this sub-group, RIP140 expression level is not correlated with good survival (right panel). Double thick blue lines represent cyto- plasmic membranes, double thin ones that of nucleus. The grey square represents GLUT3 gene, the grey ovoid forms represent GLUT3 protein. The orange circle represents glucose.
Lentiviral Particles Expressing Shrna Against Murine Glut3, supplied by Santa Cruz Biotechnology, used in various techniques. Bioz Stars score: 92/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/lentiviral particles expressing shrna against murine glut3/product/Santa Cruz Biotechnology
Average 92 stars, based on 1 article reviews
lentiviral particles expressing shrna against murine glut3 - by Bioz Stars, 2026-05
92/100 stars
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glut3 expression  (Human Protein Atlas)
90
Human Protein Atlas glut3 expression
Fig. 7 Model describing how RIP140 inhibits tumorigenesis by affecting glycolysis through the blockade of <t>GLUT3</t> expres- sion. RIP140 and p53 cooper- ate to inhibit the expression of GLUT3 induced by HIF-α. Glycolysis-dependent prolif- eration of breast cancer cells is reduced, due to a decrease in glycolysis. The prognostic value of RIP140 is associated with good survival in patients with low GLUT3, high p53 and low HIF-α (left panel). In patients with high HIF-α, low p53 and high GLUT3, RIP140 and p53 do not inhibit the tran- scriptional activity of HIF-α; GLUT3 is highly expressed and glycolysis is enhanced. In this sub-group, RIP140 expression level is not correlated with good survival (right panel). Double thick blue lines represent cyto- plasmic membranes, double thin ones that of nucleus. The grey square represents GLUT3 gene, the grey ovoid forms represent GLUT3 protein. The orange circle represents glucose.
Glut3 Expression, supplied by Human Protein Atlas, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/glut3 expression/product/Human Protein Atlas
Average 90 stars, based on 1 article reviews
glut3 expression - by Bioz Stars, 2026-05
90/100 stars
  Buy from Supplier

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Fig. 7 Model describing how RIP140 inhibits tumorigenesis by affecting glycolysis through the blockade of GLUT3 expres- sion. RIP140 and p53 cooper- ate to inhibit the expression of GLUT3 induced by HIF-α. Glycolysis-dependent prolif- eration of breast cancer cells is reduced, due to a decrease in glycolysis. The prognostic value of RIP140 is associated with good survival in patients with low GLUT3, high p53 and low HIF-α (left panel). In patients with high HIF-α, low p53 and high GLUT3, RIP140 and p53 do not inhibit the tran- scriptional activity of HIF-α; GLUT3 is highly expressed and glycolysis is enhanced. In this sub-group, RIP140 expression level is not correlated with good survival (right panel). Double thick blue lines represent cyto- plasmic membranes, double thin ones that of nucleus. The grey square represents GLUT3 gene, the grey ovoid forms represent GLUT3 protein. The orange circle represents glucose.

Journal: Cellular and molecular life sciences : CMLS

Article Title: RIP140 inhibits glycolysis-dependent proliferation of breast cancer cells by regulating GLUT3 expression through transcriptional crosstalk between hypoxia induced factor and p53.

doi: 10.1007/s00018-022-04277-3

Figure Lengend Snippet: Fig. 7 Model describing how RIP140 inhibits tumorigenesis by affecting glycolysis through the blockade of GLUT3 expres- sion. RIP140 and p53 cooper- ate to inhibit the expression of GLUT3 induced by HIF-α. Glycolysis-dependent prolif- eration of breast cancer cells is reduced, due to a decrease in glycolysis. The prognostic value of RIP140 is associated with good survival in patients with low GLUT3, high p53 and low HIF-α (left panel). In patients with high HIF-α, low p53 and high GLUT3, RIP140 and p53 do not inhibit the tran- scriptional activity of HIF-α; GLUT3 is highly expressed and glycolysis is enhanced. In this sub-group, RIP140 expression level is not correlated with good survival (right panel). Double thick blue lines represent cyto- plasmic membranes, double thin ones that of nucleus. The grey square represents GLUT3 gene, the grey ovoid forms represent GLUT3 protein. The orange circle represents glucose.

Article Snippet: Immortalized MEF#1 were transduced with lentiviral particles expressing shRNA against murine GLUT3 (sc41219-V) according to the manufacturer’s protocol (Santa Cruz Biotechnology, Dallas, USA).

Techniques: Expressing, Activity Assay